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Together, the complementary knockdown and overexpression experiments delineate a causal role for mPFC-derived CCK in the modulation of affective behaviors under social stress, providing a compelling molecular entry point for the development of targeted treatments in anxiety-related pathologies

Enzymatic Cofactor Activity : Serves as an essential cofactor for glutathione peroxidase and glutathione reductase enzymes, enabling these protective systems to neutralize hydrogen peroxide and lipid peroxides that would otherwise damage cellular membranes

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Their performance improves even further when paired with advanced treatments