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[DOI] [PMC free article] [PubMed] [Google Scholar] Hayashi H

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124 Nevertheless, if myostatin remains elevated, muscle regrowth is hampered, and if MnSOD activity is not sufficiently upregulated, unchecked oxidative stress can perpetuate tissue damage and maladaptive neurophysiological responses

Endoplasmic reticulum stress/autophagy pathway is involved in diabetes-induced neuronal apoptosis and cognitive decline in mice
